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Index by author

September 01, 2014; Volume 35,Issue 9

  1. Favaro, A.

    1. Brain
      Brain Changes in Kallmann Syndrome
      R. Manara, A. Salvalaggio, A. Favaro, V. Palumbo, V. Citton, A. Elefante, A. Brunetti, F. Di Salle, G. Bonanni, A.A. Sinisi and for the Kallmann Syndrome Neuroradiological Study Group
      American Journal of Neuroradiology September 2014, 35 (9) 1700-1706; DOI: https://doi.org/10.3174/ajnr.A3946

  2. Fiorella, D.

    1. Level 1 EBM Expedited Publication
      Open Access
      Patients Prone to Recurrence after Endovascular Treatment: Periprocedural Results of the PRET Randomized Trial on Large and Recurrent Aneurysms
      J. Raymond, R. Klink, M. Chagnon, S.L. Barnwell, A.J. Evans, J. Mocco, B.L. Hoh, A.S. Turk, R.D. Turner, H. Desal, D. Fiorella, S. Bracard, A. Weill, F. Guilbert and D. Roy on behalf of the PRET Collaborative Group
      American Journal of Neuroradiology September 2014, 35 (9) 1667-1676; DOI: https://doi.org/10.3174/ajnr.A4035

  3. Fugate, J.E.

    1. FELLOWS' JOURNAL CLUBBrain
      Early Basal Ganglia Hyperperfusion on CT Perfusion in Acute Ischemic Stroke: A Marker of Irreversible Damage?
      V. Shahi, J.E. Fugate, D.F. Kallmes and A.A. Rabinstein
      American Journal of Neuroradiology September 2014, 35 (9) 1688-1692; DOI: https://doi.org/10.3174/ajnr.A3935

      These authors found that increased cerebral blood flow and volume were seen in the basal ganglia of 4.3% of patients with ischemic strokes with CT perfusion. All patients had underlying MCA occlusions, 30% underwent hemorrhagic transformations, and the hyperperfused areas eventually became infarcted in all. Thus, acute basal ganglia hyperperfusion in patients with stroke may indicate nonviable parenchyma.

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