The Effect of Deafferentation on Cerebral Blood Flow Response to Acetazolamide

References

1. ↵
   Vorstrup S. Tomographic cerebral blood flow measurements in patients with ischemic cerebrovascular disease and evaluation of the vasodilatory capacity by the acetazolamide test. Acta Neurol Scand Suppl 1988;114:1–48

   PubMed
2. ↵
   Yonas H, Pindzola RR. Physiological determination of cerebrovascular reserves and its use in clinical management. Cerebrovasc Brain Metab Rev 1994;6:325–340

   PubMedWeb of Science
3. ↵
   Yonas H, Smith HA, Durham SR, et al. Increased stroke risk predicted by compromised cerebral blood flow reactivity. J Neurosurg 1993;79:483–489

   CrossRefPubMedWeb of Science
4. Kuroda S, Houkin K, Kamiyama H, et al. Long-term prognosis of medically treated patients with internal carotid or middle cerebral artery occlusion: can acetazolamide test predict it? Stroke 2001;32:2110–2116

   Abstract/FREE Full Text
5. ↵
   Ogasawara K, Ogawa A, Terasaki K, et al. Use of cerebrovascular reactivity in patients with symptomatic major cerebral artery occlusion to predict 5-year outcome: comparison of xenon-133 and iodine-123-IMP single-photon emission computed tomography. J Cereb Blood Flow Metab 2002;22:1142–1148

   CrossRefPubMed
6. ↵
   Nariai T, Suzuki R, Hirakawa K, et al. Vascular reserve in chronic cerebral ischemia measured by the acetazolamide challenge test: comparison with positron-emission tomography. AJNR Am J Neuroradiol 1995;16:563–570

   Abstract
7. ↵
   Yamauchi H, Okazawa H, Kishibe Y, et al. Reduced blood flow response to acetazolamide reflects pre-existing vasodilation and decreased oxygen metabolism in major cerebral arterial occlusive disease. Eur J Nucl Med 2002;29:1349–1356

   CrossRef
8. ↵
   Gotoh F, Meyer JS, Tomita M. Carbonic anhydrase inhibition and cerebral venous blood gases and ions in man. Arch Intern Med 1966;117:39–46

   CrossRefPubMedWeb of Science
9. ↵
   Garcia JH, Lassen NA, Weiller C, et al. Ischemic stroke and incomplete infarction. Stroke 1996;27:761–765

   Abstract/FREE Full Text
10. ↵
    Feeney DM, Baron JC. Diaschisis. Stroke 1986;17:817–830

    FREE Full Text
11. ↵
    Baron JC, Bousser MG, Comar D, Castaigne P. Crossed cerebellar diaschisis in human supratentorial brain infarction. Trans Am Neurol Assoc 1980;105:459–461
12. ↵
    Yamauchi H, Fukuyama H, Kimura J. Hemodynamic and metabolic changes in crossed cerebellar hypoperfusion. Stroke 1992;23:855–860

    Abstract/FREE Full Text
13. ↵
    DeGardo T, Turkington T, Williams J, et al. Performance characteristics of a whole-body PET scanner. J Nucl Med 1994;35:1398–1406

    Abstract/FREE Full Text
14. ↵
    Okazawa H, Yamauchi H, Sugimoto K, et al. Quantitative comparison of the bolus and steady-state methods for measurement of cerebral perfusion and oxygen metabolism: positron emission tomography study using ¹⁵O gas and water. J Cereb Blood Flow Metab 2001;21:793–803

    CrossRefPubMed
15. ↵
    Okazawa H, Yamauchi H, Sugimoto K, et al. Effects of acetazolamide on cerebral blood flow, blood volume, and oxygen metabolism: a positron emission tomography study with healthy volunteers. J Cereb Blood Flow Metab 2001;21:1472–1479

    CrossRefPubMedWeb of Science
16. ↵
    Votaw JR, Shulman SD. Performance evaluation of the pico-count flow-through detector for use in cerebral blood flow PET studies. J Nucl Med 1998;39:509–515

    Abstract/FREE Full Text
17. ↵
    Frackowiak RSJ, Lenzi GL, Jones T, Heather JD. Quantitative measurement of regional cerebral blood flow and oxygen metabolism in man using ¹⁵O and positron emission tomography: theory, procedure, and normal values. J Comput Assist Tomogr 1980;4:727–736

    CrossRefPubMedWeb of Science
18. ↵
    Lammertsma AA, Jones T. Correction for the presence of intravascular oxygen-15 in the steady-state technique for measuring regional oxygen extraction ratio in the brain. 1. Description of the method. J Cereb Blood Flow Metab 1983;3:416–424

    PubMedWeb of Science
19. ↵
    Herscovitch P, Markham J, Raichle ME. Brain blood flow measured with intravenous H2(15)O. I. Theory and error analysis. J Nucl Med 1983;24:782–789

    Abstract/FREE Full Text
20. ↵
    Raichle ME, Martin WRW, Herscovitch P, et al. Brain blood flow measured with intravenous H2(15)O. II. Implementation and validation. J Nucl Med 1983;24:790–798

    Abstract/FREE Full Text
21. ↵
    Bogsrud TV, Rootwelt K, Russell D, Nyberg-Hansen R. Acetazolamide effect on cerebellar blood flow in crossed cerebral-cerebellar diaschisis. Stroke 1990;21:52–55

    Abstract/FREE Full Text
22. Matsuda H, Tsuji S, Sumiya H, et al. Acetazolamide effect on vascular response in areas with diaschisis as measured by Tc-99m HMPAO brain SPECT. Clin Nucl Med 1992;17:581–586

    CrossRefPubMedWeb of Science
23. Sakashita Y, Matsuda H, Kakuda K, Takamori M. Hypoperfusion and vasoreactivity in the thalamus and cerebellum after stroke. Stroke 1993;24:84–87

    Abstract/FREE Full Text
24. ↵
    Kuwabara Y, Ichiya Y, Sasaki M, et al. Cerebellar vascular response to acetazolamide in crossed cerebellar diaschisis: a comparison of ^99mTc- HMPAO single-photon emission tomography with ¹⁵O-H2O positron emission tomography. Eur J Nucl Med 1996;23:683–689

    PubMed
25. ↵
    Ito H, Kanno I, Shimosegawa E, et al. Hemodynamic changes during neuronal deactivation in human brain: a positron emission tomography study of crossed cerebellar diaschisis. Ann Nucl Med 2002;16:249–254

    CrossRefPubMedWeb of Science
26. ↵
    Dong Y, Fukuyama H, Nabatame H, et al. Assessment of benzodiazepine receptors using iodine-123-labeled iomazenil single-photon emission computed tomography in patients with ischemic cerebrovascular disease: a comparison with PET study. Stroke 1997;28:1776–1782

    Abstract/FREE Full Text